摘要 : Toll-like receptors (TLRs), which are activated by invading microorganisms or endogenous molecules, evoke immune and inflammatory responses. TLR activation is closely linked to the development of many chronic inflammatory diseases... 展开
作者 | Youn HS Lee JY Saitoh SI Miyake K Hwang DH |
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作者单位 | |
期刊名称 | 《Biochemical and Biophysical Research Communications》 |
总页数 | 6 |
语种/中图分类号 | 英语 / Q5 |
关键词 | auranofin gold toll-like receptor LPS dimerization NF-kappa B COX-2 MyD88 TRIF IKK beta NF-KAPPA-B NECROSIS-FACTOR-ALPHA TOLL-LIKE RECEPTORS RHEUMATOID-ARTHRITIS INNATE IMMUNITY INHIBITION ACTIVATION EXPRESSION MACROPHAGES ANTAGONIST |
馆藏号 | N2007EPST0000099 |