摘要 : The proinflammatory cytokine tumor necrosis factor (TNF)-alpha has been implicated in the attenuation of neutrophil spontaneous apoptosis during sepsis. Antiapoptotic signaling is principally mediated through the p60TNF receptor (... 展开
作者 | Kilpatrick LE Lee JY Haines KM Campbell DE Sullivan KE Korchak HM |
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作者单位 | |
期刊名称 | 《American Journal of Physiology》 |
总页数 | 10 |
语种/中图分类号 | 英语 / R33 |
关键词 | 1-Phosphatidylinositol 3-Kinase Apoptosis Isoenzymes Neutrophils Protein Kinase C 1-磷脂酰肌醇3-激酶 脱噬作用 同工酶类 中性白细胞 蛋白激酶C 信号传递 |
馆藏号 | N2007EPST0000353 |